What to know
Breathing in dust from crystalline silica-containing materials can lead to lung disease. Silica causes inflammation and scarring in the lungs reducing the ability to breathe. This condition is called silicosis. Silicosis results in permanent lung damage and is a progressive, debilitating, and sometimes fatal disease.
Silicosis symptoms
Chronic silicosis typically occurs after 10 or more years of exposure to respirable crystalline silica. However, the disease can occur much more quickly after heavy exposures. Silicosis can develop or progress even after workplace exposures have stopped. Symptoms of silicosis my include:
- Cough
- Fatigue
- Shortness of breath
- Chest pain
There is no known cure for silicosis and some patients may require a lung transplant. Workers exposed to silica and those who have silicosis are also at increased risk of tuberculosis (TB). TB is a contagious and potentially life-threatening infection.
Exposure to respirable crystalline silica puts workers at risk for developing other serious diseases including:
Medical monitoring
Medical monitoring or worker surveillance can help identify health effects related to respirable crystalline silica exposure. An important concern is the risk of developing silicosis, a progressive and irreversible lung disease. Silica exposure can also impair lung function by causing COPD.
As lung tissue turns into scar tissue with silicosis, reduced lung function occurs and gets progressively worse, even after dust exposure has ended.
For a clinician to make a diagnosis of silicosis, a worker needs to have a history of exposure to respirable crystalline silica and radiographic or histopathologic findings compatible with the disease. It is also important to exclude other possible causes of lung disease. Findings of silicosis can be seen with chest x-rays, but computerized tomography (CT scans) have greater sensitivity.
Silicosis timeline
The time from initial exposure to when symptoms first appear (the latency period) depends on duration and intensity of exposure. Higher exposures result in shorter latency periods and faster disease development. The information below describes different types of silicosis.
The type with the quickest onset is acute silicosis. This can occur after a few weeks or months of exposure to very high levels of silica. Acute silicosis causes parts of the lung fill with fluid typically causing severe illness or death.
Accelerated silicosis occurs after high levels of exposure and typically presents after 5 to 10 years. Chronic silicosis is the most common type. It occurs after 10 or more years of exposure to lower levels of silica. Both types of silicosis have the same radiographic appearance and are set apart based on their different latency periods.
Periodic medical monitoring helps to detect silicosis at early stages. This is important so workers can avoid further exposure and limit disease progression. Healthcare providers who suspect a patient's health problems are caused by silica should report to their local or state health department.